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Function
Gene Ontology Displayer
27 Pathways
- Adherens junction,
- Signaling by Rho GTPases,
- MAP2K and MAPK activation,
- RAF/MAP kinase cascade,
- Cell-Cell communication,
- Regulation of actin cytoskeleton,
- Paradoxical activation of RAF signaling by kinase inactive BRAF,
- Signaling by BRAF and RAF fusions,
- Regulation of insulin secretion,
- Metabolism,
- Integration of energy metabolism,
- Signaling by RAS mutants,
- MAPK1/MAPK3 signaling,
- MAPK family signaling cascades,
- RHO GTPases activate IQGAPs,
- Glucagon-like Peptide-1 (GLP1) regulates insulin secretion,
- Immune System,
- Diseases of signal transduction,
- Disease,
- Innate Immune System,
- Signal Transduction,
- Nephrin family interactions,
- Neutrophil degranulation,
- Signaling by moderate kinase activity BRAF mutants,
- Signaling by high-kinase activity BRAF mutants,
- Oncogenic MAPK signaling,
- RHO GTPase Effectors
Gene --> GO terms.
Gene -> HPO annotation (Human Phenotype Ontology)
Mouse Gene --> Allele [Phenotype]
Genomics
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Gene Structure Displayer
Overlapping Features Displayer
Mouse Alleles Displayer
Gene --> Chromosomal location.
Gene --> GO terms.
Gene --> Overlapping genes.
Disease
Gene (Hum OR Rat) --> Mouse Allele (Phenotype)
Gene --> Alleles and Disease (clinVar data)
Gene -> HPO annotation (Human Phenotype Ontology)
Mouse Gene --> Allele [Phenotype]
Other
27 Pathways
- Adherens junction,
- Signaling by Rho GTPases,
- MAP2K and MAPK activation,
- RAF/MAP kinase cascade,
- Cell-Cell communication,
- Regulation of actin cytoskeleton,
- Paradoxical activation of RAF signaling by kinase inactive BRAF,
- Signaling by BRAF and RAF fusions,
- Regulation of insulin secretion,
- Metabolism,
- Integration of energy metabolism,
- Signaling by RAS mutants,
- MAPK1/MAPK3 signaling,
- MAPK family signaling cascades,
- RHO GTPases activate IQGAPs,
- Glucagon-like Peptide-1 (GLP1) regulates insulin secretion,
- Immune System,
- Diseases of signal transduction,
- Disease,
- Innate Immune System,
- Signal Transduction,
- Nephrin family interactions,
- Neutrophil degranulation,
- Signaling by moderate kinase activity BRAF mutants,
- Signaling by high-kinase activity BRAF mutants,
- Oncogenic MAPK signaling,
- RHO GTPase Effectors
