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Function
Gene Ontology Displayer
27 Pathways
- G1 Phase,
- Diseases of mitotic cell cycle,
- Senescence-Associated Secretory Phenotype (SASP),
- Oncogene Induced Senescence,
- Regulation of RUNX1 Expression and Activity,
- Mitotic G1 phase and G1/S transition,
- Transcriptional regulation by RUNX1,
- RNA Polymerase II Transcription,
- Defective binding of RB1 mutants to E2F1,(E2F2, E2F3),
- Diseases of cellular response to stress,
- Evasion of Oxidative Stress Induced Senescence Due to p16INK4A Defects,
- Gene expression (Transcription),
- Evasion of Oxidative Stress Induced Senescence Due to Defective p16INK4A binding to CDK4 and CDK6,
- Cellular responses to stress,
- Disease,
- Cell Cycle, Mitotic,
- Diseases of Cellular Senescence,
- Evasion of Oncogene Induced Senescence Due to Defective p16INK4A binding to CDK4 and CDK6,
- Evasion of Oncogene Induced Senescence Due to p16INK4A Defects,
- Cellular Senescence,
- Generic Transcription Pathway,
- Cyclin D associated events in G1,
- Cell Cycle,
- Aberrant regulation of mitotic cell cycle due to RB1 defects,
- Cellular responses to external stimuli,
- Oxidative Stress Induced Senescence,
- Aberrant regulation of mitotic G1/S transition in cancer due to RB1 defects
Gene --> GO terms.
Gene -> HPO annotation (Human Phenotype Ontology)
Mouse Gene --> Allele [Phenotype]
Genomics
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Overlapping Features Displayer
Mouse Alleles Displayer
Gene --> Chromosomal location.
Gene --> GO terms.
Gene --> Overlapping genes.
Disease
Gene (Hum OR Rat) --> Mouse Allele (Phenotype)
Gene --> Alleles and Disease (clinVar data)
Gene -> HPO annotation (Human Phenotype Ontology)
Mouse Gene --> Allele [Phenotype]
Other
27 Pathways
- G1 Phase,
- Diseases of mitotic cell cycle,
- Senescence-Associated Secretory Phenotype (SASP),
- Oncogene Induced Senescence,
- Regulation of RUNX1 Expression and Activity,
- Mitotic G1 phase and G1/S transition,
- Transcriptional regulation by RUNX1,
- RNA Polymerase II Transcription,
- Defective binding of RB1 mutants to E2F1,(E2F2, E2F3),
- Diseases of cellular response to stress,
- Evasion of Oxidative Stress Induced Senescence Due to p16INK4A Defects,
- Gene expression (Transcription),
- Evasion of Oxidative Stress Induced Senescence Due to Defective p16INK4A binding to CDK4 and CDK6,
- Cellular responses to stress,
- Disease,
- Cell Cycle, Mitotic,
- Diseases of Cellular Senescence,
- Evasion of Oncogene Induced Senescence Due to Defective p16INK4A binding to CDK4 and CDK6,
- Evasion of Oncogene Induced Senescence Due to p16INK4A Defects,
- Cellular Senescence,
- Generic Transcription Pathway,
- Cyclin D associated events in G1,
- Cell Cycle,
- Aberrant regulation of mitotic cell cycle due to RB1 defects,
- Cellular responses to external stimuli,
- Oxidative Stress Induced Senescence,
- Aberrant regulation of mitotic G1/S transition in cancer due to RB1 defects
