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Function
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49 Pathways
- p53-Dependent G1 DNA Damage Response,
- Transcriptional Regulation by TP53,
- Metabolism of proteins,
- RNA Polymerase II Transcription,
- Cellular Senescence,
- Generic Transcription Pathway,
- Gene expression (Transcription),
- SUMOylation of transcription factors,
- SUMO E3 ligases SUMOylate target proteins,
- Transcriptional Regulation by VENTX,
- Mitotic G1 phase and G1/S transition,
- p53-Dependent G1/S DNA damage checkpoint,
- Regulation of RUNX3 expression and activity,
- SUMOylation,
- Cell Cycle,
- Diseases of programmed cell death,
- Cellular responses to external stimuli,
- Regulation of TP53 Expression and Degradation,
- G1 Phase,
- Evasion of Oxidative Stress Induced Senescence Due to p14ARF Defects,
- Defective Intrinsic Pathway for Apoptosis Due to p14ARF Loss of Function,
- Stabilization of p53,
- Diseases of cellular response to stress,
- Senescence-Associated Secretory Phenotype (SASP),
- Transcriptional regulation by RUNX3,
- Evasion of Oxidative Stress Induced Senescence Due to Defective p16INK4A binding to CDK4 and CDK6,
- Cyclin D associated events in G1,
- Regulation of TP53 Degradation,
- Oxidative Stress Induced Senescence,
- Evasion of Oncogene Induced Senescence Due to Defective p16INK4A binding to CDK4,
- Evasion of Oxidative Stress Induced Senescence Due to p16INK4A Defects,
- Apoptosis,
- SUMOylation of DNA damage response and repair proteins,
- Oncogene Induced Senescence,
- Evasion of Oncogene Induced Senescence Due to Defective p16INK4A binding to CDK4 and CDK6,
- Cell Cycle Checkpoints,
- Programmed Cell Death,
- Evasion of Oncogene Induced Senescence Due to p16INK4A Defects,
- Evasion of Oxidative Stress Induced Senescence Due to Defective p16INK4A binding to CDK4,
- Regulation of TP53 Activity,
- Diseases of Cellular Senescence,
- Cellular responses to stress,
- Intrinsic Pathway for Apoptosis,
- Evasion of Oncogene Induced Senescence Due to p14ARF Defects,
- Apoptotic factor-mediated response,
- Cell Cycle, Mitotic,
- G1/S DNA Damage Checkpoints,
- Disease,
- Post-translational protein modification
Gene --> GO terms.
Gene -> HPO annotation (Human Phenotype Ontology)
Mouse Gene --> Allele [Phenotype]
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Gene --> Chromosomal location.
Gene --> GO terms.
Gene --> Overlapping genes.
Disease
Gene (Hum OR Rat) --> Mouse Allele (Phenotype)
Gene --> Alleles and Disease (clinVar data)
Gene -> HPO annotation (Human Phenotype Ontology)
Mouse Gene --> Allele [Phenotype]
Other
49 Pathways
- p53-Dependent G1 DNA Damage Response,
- Transcriptional Regulation by TP53,
- Metabolism of proteins,
- RNA Polymerase II Transcription,
- Cellular Senescence,
- Generic Transcription Pathway,
- Gene expression (Transcription),
- SUMOylation of transcription factors,
- SUMO E3 ligases SUMOylate target proteins,
- Transcriptional Regulation by VENTX,
- Mitotic G1 phase and G1/S transition,
- p53-Dependent G1/S DNA damage checkpoint,
- Regulation of RUNX3 expression and activity,
- SUMOylation,
- Cell Cycle,
- Diseases of programmed cell death,
- Cellular responses to external stimuli,
- Regulation of TP53 Expression and Degradation,
- G1 Phase,
- Evasion of Oxidative Stress Induced Senescence Due to p14ARF Defects,
- Defective Intrinsic Pathway for Apoptosis Due to p14ARF Loss of Function,
- Stabilization of p53,
- Diseases of cellular response to stress,
- Senescence-Associated Secretory Phenotype (SASP),
- Transcriptional regulation by RUNX3,
- Evasion of Oxidative Stress Induced Senescence Due to Defective p16INK4A binding to CDK4 and CDK6,
- Cyclin D associated events in G1,
- Regulation of TP53 Degradation,
- Oxidative Stress Induced Senescence,
- Evasion of Oncogene Induced Senescence Due to Defective p16INK4A binding to CDK4,
- Evasion of Oxidative Stress Induced Senescence Due to p16INK4A Defects,
- Apoptosis,
- SUMOylation of DNA damage response and repair proteins,
- Oncogene Induced Senescence,
- Evasion of Oncogene Induced Senescence Due to Defective p16INK4A binding to CDK4 and CDK6,
- Cell Cycle Checkpoints,
- Programmed Cell Death,
- Evasion of Oncogene Induced Senescence Due to p16INK4A Defects,
- Evasion of Oxidative Stress Induced Senescence Due to Defective p16INK4A binding to CDK4,
- Regulation of TP53 Activity,
- Diseases of Cellular Senescence,
- Cellular responses to stress,
- Intrinsic Pathway for Apoptosis,
- Evasion of Oncogene Induced Senescence Due to p14ARF Defects,
- Apoptotic factor-mediated response,
- Cell Cycle, Mitotic,
- G1/S DNA Damage Checkpoints,
- Disease,
- Post-translational protein modification
