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Function
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22 Pathways
- Post-translational protein modification,
- Removal of aminoterminal propeptides from gamma-carboxylated proteins,
- Defective cofactor function of FVIIIa variant,
- Defective factor IX causes thrombophilia,
- Extrinsic Pathway of Fibrin Clot Formation,
- Gamma carboxylation, hypusine formation and arylsulfatase activation,
- Metabolism of proteins,
- Hemostasis,
- Defective F9 variant does not activate FX,
- Defects of contact activation system (CAS) and kallikrein/kinin system (KKS),
- Defective factor VIII causes hemophilia A,
- Defective F9 secretion,
- Gamma-carboxylation, transport, and amino-terminal cleavage of proteins,
- Defective factor IX causes hemophilia B,
- Diseases of hemostasis,
- Transport of gamma-carboxylated protein precursors from the endoplasmic reticulum to the Golgi apparatus,
- Intrinsic Pathway of Fibrin Clot Formation,
- Gamma-carboxylation of protein precursors,
- Defective F9 activation,
- Disease,
- Defective gamma-carboxylation of F9,
- Formation of Fibrin Clot (Clotting Cascade)
Gene --> GO terms.
Gene -> HPO annotation (Human Phenotype Ontology)
Mouse Gene --> Allele [Phenotype]
Genomics
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Gene --> Chromosomal location.
Gene --> GO terms.
Gene --> Overlapping genes.
Disease
Gene (Hum OR Rat) --> Mouse Allele (Phenotype)
Gene --> Alleles and Disease (clinVar data)
Gene -> HPO annotation (Human Phenotype Ontology)
Mouse Gene --> Allele [Phenotype]
Other
22 Pathways
- Post-translational protein modification,
- Removal of aminoterminal propeptides from gamma-carboxylated proteins,
- Defective cofactor function of FVIIIa variant,
- Defective factor IX causes thrombophilia,
- Extrinsic Pathway of Fibrin Clot Formation,
- Gamma carboxylation, hypusine formation and arylsulfatase activation,
- Metabolism of proteins,
- Hemostasis,
- Defective F9 variant does not activate FX,
- Defects of contact activation system (CAS) and kallikrein/kinin system (KKS),
- Defective factor VIII causes hemophilia A,
- Defective F9 secretion,
- Gamma-carboxylation, transport, and amino-terminal cleavage of proteins,
- Defective factor IX causes hemophilia B,
- Diseases of hemostasis,
- Transport of gamma-carboxylated protein precursors from the endoplasmic reticulum to the Golgi apparatus,
- Intrinsic Pathway of Fibrin Clot Formation,
- Gamma-carboxylation of protein precursors,
- Defective F9 activation,
- Disease,
- Defective gamma-carboxylation of F9,
- Formation of Fibrin Clot (Clotting Cascade)