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Function
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61 Pathways
- Interferon Signaling,
- RNA Polymerase II Transcription,
- CREB1 phosphorylation through NMDA receptor-mediated activation of RAS signaling,
- Calmodulin induced events,
- Disease,
- Immune System,
- RAF/MAP kinase cascade,
- G alpha (i) signalling events,
- Gene expression (Transcription),
- Negative regulation of NMDA receptor-mediated neuronal transmission,
- Opioid Signalling,
- MAPK family signaling cascades,
- Paradoxical activation of RAF signaling by kinase inactive BRAF,
- Diseases of signal transduction by growth factor receptors and second messengers,
- Unblocking of NMDA receptors, glutamate binding and activation,
- Phase 0 - rapid depolarisation,
- Trafficking of AMPA receptors,
- Ion transport by P-type ATPases,
- Signaling downstream of RAS mutants,
- CaM pathway,
- CaMK IV-mediated phosphorylation of CREB,
- RAF activation,
- Ion homeostasis,
- Post NMDA receptor activation events,
- Long-term potentiation,
- Assembly and cell surface presentation of NMDA receptors,
- Ion channel transport,
- Intracellular signaling by second messengers,
- Glutamate binding, activation of AMPA receptors and synaptic plasticity,
- Signaling by RAS mutants,
- DAG and IP3 signaling,
- Muscle contraction,
- Neurotransmitter receptors and postsynaptic signal transmission,
- MAPK1/MAPK3 signaling,
- Transcriptional Regulation by MECP2,
- Transmission across Chemical Synapses,
- Cellular responses to external stimuli,
- Cytokine Signaling in Immune system,
- FLT3 Signaling,
- Cardiac conduction,
- G-protein mediated events,
- Signaling by RAF1 mutants,
- Signal Transduction,
- Oncogenic MAPK signaling,
- Signaling by moderate kinase activity BRAF mutants,
- Ras activation upon Ca2+ influx through NMDA receptor,
- Neuronal System,
- Transport of small molecules,
- Interferon gamma signaling,
- CREB1 phosphorylation through the activation of CaMKII/CaMKK/CaMKIV cascasde,
- Cellular responses to stress,
- Activation of NMDA receptors and postsynaptic events,
- Regulation of MECP2 expression and activity,
- Signaling by GPCR,
- Generic Transcription Pathway,
- GPCR downstream signalling,
- Signaling by BRAF and RAF fusions,
- Ca-dependent events,
- Cellular response to heat stress,
- HSF1-dependent transactivation,
- PLC beta mediated events
Gene --> GO terms.
Gene -> HPO annotation (Human Phenotype Ontology)
Mouse Gene --> Allele [Phenotype]
Genomics
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Disease
Gene (Hum OR Rat) --> Mouse Allele (Phenotype)
Gene --> Alleles and Disease (clinVar data)
Gene -> HPO annotation (Human Phenotype Ontology)
Mouse Gene --> Allele [Phenotype]
Other
61 Pathways
- Interferon Signaling,
- RNA Polymerase II Transcription,
- CREB1 phosphorylation through NMDA receptor-mediated activation of RAS signaling,
- Calmodulin induced events,
- Disease,
- Immune System,
- RAF/MAP kinase cascade,
- G alpha (i) signalling events,
- Gene expression (Transcription),
- Negative regulation of NMDA receptor-mediated neuronal transmission,
- Opioid Signalling,
- MAPK family signaling cascades,
- Paradoxical activation of RAF signaling by kinase inactive BRAF,
- Diseases of signal transduction by growth factor receptors and second messengers,
- Unblocking of NMDA receptors, glutamate binding and activation,
- Phase 0 - rapid depolarisation,
- Trafficking of AMPA receptors,
- Ion transport by P-type ATPases,
- Signaling downstream of RAS mutants,
- CaM pathway,
- CaMK IV-mediated phosphorylation of CREB,
- RAF activation,
- Ion homeostasis,
- Post NMDA receptor activation events,
- Long-term potentiation,
- Assembly and cell surface presentation of NMDA receptors,
- Ion channel transport,
- Intracellular signaling by second messengers,
- Glutamate binding, activation of AMPA receptors and synaptic plasticity,
- Signaling by RAS mutants,
- DAG and IP3 signaling,
- Muscle contraction,
- Neurotransmitter receptors and postsynaptic signal transmission,
- MAPK1/MAPK3 signaling,
- Transcriptional Regulation by MECP2,
- Transmission across Chemical Synapses,
- Cellular responses to external stimuli,
- Cytokine Signaling in Immune system,
- FLT3 Signaling,
- Cardiac conduction,
- G-protein mediated events,
- Signaling by RAF1 mutants,
- Signal Transduction,
- Oncogenic MAPK signaling,
- Signaling by moderate kinase activity BRAF mutants,
- Ras activation upon Ca2+ influx through NMDA receptor,
- Neuronal System,
- Transport of small molecules,
- Interferon gamma signaling,
- CREB1 phosphorylation through the activation of CaMKII/CaMKK/CaMKIV cascasde,
- Cellular responses to stress,
- Activation of NMDA receptors and postsynaptic events,
- Regulation of MECP2 expression and activity,
- Signaling by GPCR,
- Generic Transcription Pathway,
- GPCR downstream signalling,
- Signaling by BRAF and RAF fusions,
- Ca-dependent events,
- Cellular response to heat stress,
- HSF1-dependent transactivation,
- PLC beta mediated events
