Uni Prot Comments Displayer
Protein Atlas Displayer
Gene Expression Atlas Tissues Displayer
Gene Expression Atlas Diseases Displayer
Publication Counts Displayer
Curated Proteins Displayer
Function
Gene Ontology Displayer
24 Pathways
- Signal Transduction,
- Nucleotide-like (purinergic) receptors,
- Infectious disease,
- G alpha (s) signalling events,
- Leishmania parasite growth and survival,
- Class A/1 (Rhodopsin-like receptors),
- Signaling by GPCR,
- Metabolism of proteins,
- Signaling by NTRK1 (TRKA),
- Calcium signaling pathway,
- Leishmania infection,
- Vascular smooth muscle contraction,
- Signaling by Receptor Tyrosine Kinases,
- Activation of TRKA receptors,
- Surfactant metabolism,
- Disease,
- ADORA2B mediated anti-inflammatory cytokines production,
- GPCR ligand binding,
- Neuroactive ligand-receptor interaction,
- NGF-independant TRKA activation,
- GPCR downstream signalling,
- Anti-inflammatory response favouring Leishmania parasite infection,
- Adenosine P1 receptors,
- Signaling by NTRKs
Gene --> GO terms.
Gene -> HPO annotation (Human Phenotype Ontology)
Mouse Gene --> Allele [Phenotype]
Genomics
J Browse Displayer
Gene Structure Displayer
Overlapping Features Displayer
Mouse Alleles Displayer
Gene --> Chromosomal location.
Gene --> GO terms.
Gene --> Overlapping genes.
Disease
Gene (Hum OR Rat) --> Mouse Allele (Phenotype)
Gene --> Alleles and Disease (clinVar data)
Gene -> HPO annotation (Human Phenotype Ontology)
Mouse Gene --> Allele [Phenotype]
Other
24 Pathways
- Signal Transduction,
- Nucleotide-like (purinergic) receptors,
- Infectious disease,
- G alpha (s) signalling events,
- Leishmania parasite growth and survival,
- Class A/1 (Rhodopsin-like receptors),
- Signaling by GPCR,
- Metabolism of proteins,
- Signaling by NTRK1 (TRKA),
- Calcium signaling pathway,
- Leishmania infection,
- Vascular smooth muscle contraction,
- Signaling by Receptor Tyrosine Kinases,
- Activation of TRKA receptors,
- Surfactant metabolism,
- Disease,
- ADORA2B mediated anti-inflammatory cytokines production,
- GPCR ligand binding,
- Neuroactive ligand-receptor interaction,
- NGF-independant TRKA activation,
- GPCR downstream signalling,
- Anti-inflammatory response favouring Leishmania parasite infection,
- Adenosine P1 receptors,
- Signaling by NTRKs
